Notably, Single Nucleotide Polymorphism (SNP) analyses have revealed connections between psoriasis and genes related to Th2 (e.g., IL-4, IL-13), Th17 (e.g., IL-12, IL-23), innate immune responses (e.g., Nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB), interferons (IFNs), and cluster of differentiation 8 (CD8)+ T cell-mediated adaptive immunity [62]. The gene discussed is IL37; the disease is psoriasis.