IFNA1 and autism: Our previous RNA‐seq analysis of choroid plexus from adult offspring of PBS and MIA mice revealed significant enrichment of antiviral and IFN signaling pathways, and both qPCR and serum assays confirmed the most pronounced change in IFN‐γ; furthermore, blocking the IFN‐γ receptor markedly alleviated autism‐like behaviors and ventricular enlargement in MIA offspring, indicating that IFN‐γ signaling plays a critical role in the pathological process [7].