ZFPL1 and colorectal carcinoma: Conversely, overexpression of both ZFPL1 and its transmembrane domain‐deficient variant (ZFPL1ΔTMD) promoted cellular proliferation and activated p‐Akt308 and p‐Akt473 expressions.[29] Despite these advancements, the precise role of ZFPL1 in CRC pathogenesis remains poorly understood.