While we observed repression of a handful genes involved in cardiomyocyte differentiation through BMP axis (BMPR1B) [49], survival (BIRC3) [50] sarcomere organization (RAC2) [51], and hypertension (ADRA2) [52, 53]; in YAP1-KO CMs, we did not observe binding to previously described non-CM targets such as CTGF, CYR61, and AXL or to CM specific hypertrophy associated targets containing TEAD consensus sequence such as MYH7 or NFAT. This evidence concerns the gene CCN2 and hypertensive disorder.