To this list, we added two genes from previous CAD GWAS studies (RBPMS2 and TCF21) and two genes with rare-mutation evidence in the FinnGen database (HHIPL1 and ADAMTS7), which had supporting evidence for SMC-mediated function based on previous studies,5,11,17,18,19,20 but were not detected in our pipeline for various reasons, e.g., low expression in scRNA-seq data. This evidence concerns the gene HHIPL1 and coronary artery disorder.