MIF and lung adenocarcinoma: Interestingly, intersecting the previously identified glycolysis-related genes with the marker genes of alveolar epithelial cells revealed that MIF can simultaneously serve as a differential gene in the glycolysis pathway and a marker gene for alveolar epithelial cells (Figure 2D), indicating that MIF is a key gene involved in regulating glycolysis in driver gene-negative lung adenocarcinoma.