Identified a RORγt+ Thy1hiSCA-1+ ILC subset that produces IL - 17 and IFN-γ under IL - 23 stimulation and mediates innate immune colitis in mouse models. Suggests ILCs share functional features with Th17 cells in IL - 23-driven inflammation and may be therapeutic targets in diseases like psoriasis. Here, IFNG is linked to colitis.