AKT1 and Sepsis: Huang et al. [8] reported that ANXA1 can induce the differentiation of CD4 T cells into Th0/Th1 and improve sepsis via immunosuppression by regulating GATA‐3 and T‐bet molecules through the extracellular regulated protein kinases (ERK)/protein kinase B (PKB/Akt) pathway activation.