demonstrated that exposure to IFN‐I early during infection with recombinant vesicular stomatitis virus (VSV) induces IL‐6 production from migratory cDC2s and mo‐DCs and promotes Tfh cell priming, whereas late IFN‐I exposure during infection with LCMV is associated with low IL‐6 production and favors non‐Tfh (Th1) differentiation [196]. Here, IL6 is linked to infection.