In terms anti-inflammatory pathways, FMN significantly decreases inflammatory cell infiltration in bronchoalveolar lavage fluid (BALF), increases superoxide dismutase (SOD) activity, and inhibits myeloperoxidase (MPO) activity in lipopolysaccharide (LPS)-induced mouse models of ALI (Ma et al., 2013). The gene discussed is SOD1; the disease is acute respiratory distress syndrome.