Functional enrichment analysis demonstrated that the complex interaction network of protein products of upregulated FBLN1, EFEMP1, and ANTXR1 genes in the EC of female AD patients compared to female controls was mainly involved in biological processes related to positive regulation of substrate-dependent cell migration, negative regulation of transforming growth factor beta (TGFβ) production, toxin transport, and regulation of ERK1 and ERK2 cascade (Appendix H) [53,54]. The gene discussed is TGFB1; the disease is Alzheimer disease.