Some mutations of PARK2, LRRK2, glucosylceramidase beta (GBA), PINK1, and SNCA are among the top genetic variants increasing the risk for fPD, whereas inositol polyphosphate-5-phosphatase F (INPP5F), microtubule-associated protein tau (MAPT), and KAT8 regulatory NSL complex subunit 1 (KANSL1) with single-nucleotide polymorphisms (SNPs) constitute the top three polymorphic genes enhancing the risk for developing sporadic PD (sPD) [169]. This evidence concerns the gene KANSL1 and Platelet storage pool disease.