NFKB1 and Behcet disease: The mechanism of action of BD in the treatment of UC may be based on the substance of β-sitosterol, stigmasterol, quercetin, isorhamnetin, berberine, and anemoside B4, which act on the NF-κB signaling pathway to achieve anti-inflammatory effects and exert therapeutic effects by regulating the secretion of cytokines, modulating the expression of p65 and IκBα proteins, and inhibiting their phosphorylation.