Chemotactic interactions promote attraction and activation of cytotoxic cell subsets, likely promoting anti-tumor activity in the TME of R (pDC GPI → NK AMFR, pDC MIF → NK T CD74/CXCR4, and NK T CCL3L1 → CD8 + T CCR5) [86–88]. The gene discussed is CCR5; the disease is neoplasm.