DOCK2 and psoriasis: Notably, Sult2b1−/−Dock2−/− mice exhibited significant recovery of psoriasis-like phenotypes and neutrophil infiltration in the skin compared to Sult2b1−/−Dock2+/+ mice (Figures 4K, L), suggesting that CS alleviates IMQ-induced psoriatic dermatitis by suppressing DOCK2 function.