IL17A and neoplasm: MAIT cells in MM and AML patients highly express inhibitory receptors such as PD‐1 and TIM‐3, and cytokines such as IL‐10 and TGF‐β can weaken their cytokine secretion ability, or even make them switch to a proinflammatory phenotype that secretes IL‐17A, thereby promoting tumor angiogenesis [213, 223].