To further explore the potential role of specific myeloid subpopulations in LEMS invasion and metastases, we performed sub‐clustering analysis of myeloid cells, identifying nine distinct clusters: classical monocytes, folate receptor 2 (FOLR2+) macrophages, pro‐inflammatory monocytes, proliferating macrophages, SPP1+ macrophages, DC1, DC2, neutrophils, and plasmacytoid DC (Figure 5D–F). This evidence concerns the gene SPP1 and Lambert-Eaton myasthenic syndrome.