Further, UNC2025 strongly suppressed prosurvival signaling, triggered apoptosis, and markedly reduced proliferation and colony formation in MERTK-expressing acute lymphoblastic leukemias (ALL), acute myeloid leukemias (AML), melanoma cell lines, as well as patient-derived samples in preclinical trials [38,39]. Here, MERTK is linked to acute myeloid leukemia.