conducted research on human AT-MSCs from healthy and T2D donors, which were purchased from the same source as in the current study (Lonza), and demonstrated decreased expression of analyzed adipogenesis-related genes (CEBPα, FAS, FABP4, DGAT1, SCD-1, SREBP-1c), transcription factors (PPARα, PPARδ, PPARγ) and parallel lower accumulation of lipid droplets in diabetic AT-MSCs at 21 days of adipogenic stimulation when compared to healthy donor-derived AT-MSCs, which is consistent with our observations. The gene discussed is PPARG; the disease is type 2 diabetes mellitus.