We speculated that the fusion of KIAA0319L and PARK7 results in a protein that integrates the endocytosis capabilities of KIAA0319L [24] with PARK7, an enzyme mutated in hereditary Parkinson’s disease, [25] potentially influencing SP-EPN_6 cellular responses to oxidative stress (Supplementary Fig. 4A). This evidence concerns the gene PARK7 and Parkinson disease.