CD4 and psoriasis: These data demonstratethat GS treatment interferes with the activationof DCs in vivo and the differentiation and activation of CD4+ T lymphocytes, which may explain the prolongation of graft survival.Considering other inflammatory processes related to the skin, GS wasefficient in the treatment of mice in a psoriasis model induced byIMQ, reducing the proportions of Th1 and Th17 cells, and in lupus-likeMRL/Lpr mice, increasing life expectancy and decreasing the proportionsof plasma cells, follicular T cells, neutrophils, and DCs dLNs.