CDKN1A and lung carcinoma: Although miR‐451, another RBC‐derived miRNA, was also transferred to lung cancer cells via exosomes, it did not promote tumorigenic phenotypes, most likely due to its well‐established role as a housekeeping miRNA in RBCs, primarily involved in erythroid homeostasis.[39] This contrast underscores the specificity of miR‐93‐5p in driving lung cancer progression.[40] In addition to PTEN, our analysis identified other tumor suppressor genes, including TP53INP1, THBS2, and CDKN1A, as potential targets of miR‐93‐5p, suggesting broader regulatory roles in tumorigenic pathways.