Although miR‐451, another RBC‐derived miRNA, was also transferred to lung cancer cells via exosomes, it did not promote tumorigenic phenotypes, most likely due to its well‐established role as a housekeeping miRNA in RBCs, primarily involved in erythroid homeostasis.[39] This contrast underscores the specificity of miR‐93‐5p in driving lung cancer progression.[40] In addition to PTEN, our analysis identified other tumor suppressor genes, including TP53INP1, THBS2, and CDKN1A, as potential targets of miR‐93‐5p, suggesting broader regulatory roles in tumorigenic pathways. The gene discussed is PTEN; the disease is lung cancer.