METTL3 overexpression enhances tumor cell viability, migration, and invasion through Bcl-2 pathway modulation and facilitates metastasis through c-MYC-mediated stabilization of m6A-enriched LINC01006 and increased translation of oncogenes, including EGFR and BRD4—demonstrated in both in vitro and in vivo models [30,31]. Here, EGFR is linked to neoplasm.