Based on our previous findings demonstrating that inhibitors of glycolysis and glutaminolysis, as well as of MCTs, known to be involved in the export of lactate out of host cells, inhibited C. parvum infection in vitro [12], we wondered whether an inhibition of mTOR, as one of the master regulators of glycolysis and glutaminolysis, might also be a suitable therapeutic target for the treatment of cryptosporidiosis. The gene discussed is MTOR; the disease is cryptosporidiosis.