In conclusion, our study demonstrates that the CXG solution exerts multi-faceted anti-glioblastoma effects by simultaneously inducing apoptosis through the hsa-miR-10a-5p/BCL2L11/BCL2 axis, suppressing extracellular matrix organization, and disrupting lipid metabolism via the SREBF1/E2F1/hsa-miR-29a-3p regulatory network. The gene discussed is BCL2L11; the disease is glioblastoma.