The tetrapeptide AaTs-1, isolated from the venom of Androctonus australis, acts as a selective antagonist of the formyl-peptide receptor-like 1 (FPRL-1) in glioblastoma cells, leading to increased p53 expression while suppressing ERK/p38/JNK signaling [117]. The gene discussed is FPR2; the disease is glioblastoma.