As a result of these events, numerous pyrethroid-resistant malaria vectors in Africa have been found to exhibit high levels of P450 activity, thus decreasing the efficiency of malaria control measures by vectors that include enzymes that increase resistance to pyrethroids and carbamates, such as CYP6M2 and CYP6P3 in Anopheles gambiae and CYP6P9a and CYP6P9b in Anopheles funestus [6,7]. This evidence concerns the gene CYP2B6 and malaria.