Since sleep and circadian disruptions influence neurodegenerative diseases [24,36,37], we sought to determine whether FABP7 plays a role in AD pathophysiology and found that Aβ-induced sleep fragmentation in an Aβ fly model was rescued by overexpression of mouse FABP7 or the fly homolog, dFABP [93]. The gene discussed is FABP7; the disease is neurodegenerative disease.