KLK15 and Disorder of lipid metabolism: R7I enhanced the transcription of FABP1 and ACO and inhibited the level of SCD-1, while the effect was inhibited by treatment with GW9662 (PPARγ inhibitor), indicating that R7I regulated fatty acid (FA) transport [55] and the lipolytic [56] and FA synthesis [57] disorder caused by Neisseria infection Via PPARγ; it then regulated lipid metabolism disorder and the distribution of lipid droplets.