LDLR and hypertriglyceridemia: Numerous genome-wide association studies (GWASs) and whole-exome sequencing (WES) analyses have identified several genes such as Apolipoprotein A5 (APOA5), Lipoprotein lipase (LPL), Apolipoprotein C2 (APOC2), Familial lipase maturation factor 1 (LMF1), and Glycosylphosphatidylinositol-Anchored High-Density (GPIHBP1), variants of which lead to hypertriglyceridemia [20,21,22], while variants of Apolipoprotein B, (APOB), Low-density lipoprotein receptor (LDLR), and Proprotein Convertase Subtilisin/Kexin Type 9 (PCSK9) lead to hypercholesteremia [23,24,25,26].