Another study on cervical cancer described that the accumulation of immunoevasive lipid-laden tumor-infiltrating dendritic cells (TIDCs) was mediated by PPAR-α, and a combination of the PPAR-α antagonist GW6471 with a PD-L1 blocking antibody led to decreased tumor burden and increased survival in mice [181]. The gene discussed is PPARA; the disease is neoplasm.