The persistent fibrosis observed in OGC+/− mice aligns with previous reports of significantly lower GSH levels in idiopathic pulmonary fibrosis (Beeh et al. 2002), and liver fibrosis (Fraser et al. 2022; Huang et al. 2023), suggesting a potential role for GSH in this process. This evidence concerns the gene SLC25A11 and pulmonary fibrosis.