For subjects that had no brain fog but had other neurological symptoms, from headache, dizziness, dysgeusia, and anosmia, myalgia, numbness/tingling, or pain other than in the chest, high C5a (in 100 %) and high Gliomedin (in 50 %) appeared to be more closely associated in these subjects, in contrast to TGF-β1 which was normal in any of such subjects (Table 8). Here, C5 is linked to Kallmann syndrome.