While ZNFs are known for mitochondrial protein import [80], brain development [81, 82], and some links to AD pathology [54], our innovation is the eQTL analysis exposing significant interactions between these SNPs and the mitochondrial epistatic gene CEACAM19 (itself linked to AD risk via TWAS [83]), suggesting novel co‐regulatory mechanisms involving our identified genetic markers, CEACAM19, and established AD genes. Here, CEACAM19 is linked to Alzheimer disease.