In addition, MoS2 exposure-induced increase of serum ALT and AST levels, as well as the pathological changes determined by H&E staining, were all reversed when pretreatment with BFA or XMU-XP-1 (Fig. 8C–E), indicating that MoS2-induced acute liver injury was also through MST2/LC3B axis. The gene discussed is STK3; the disease is injury.