The renal phenotypes linked to these WDR19 variants are not well-characterized, showing diverse histological abnormalities, including interstitial fibrosis, atrophic tubules, glomerular sclerosis, glomerular cysts, and rarely, evidence of IgA deposits, which may mimic immune complex–mediated glomerulopathy, complicating the understanding of disease etiology.13 This evidence concerns the gene WDR19 and glomerulosclerosis.