Specifically, C1orf122 promoted HCC cell growth via apoptosis inhibition, as indicated by its significant effect on the expression of apoptotic pathway-related genes such as Bax, Bcl-2, Phospho-Bcl-2, Caspase-3, and Cleaved-Caspase-3, among others. The gene discussed is BAX; the disease is hepatocellular carcinoma.