Specifically, C1orf122 promoted HCC cell growth via apoptosis inhibition, as indicated by its significant effect on the expression of apoptotic pathway-related genes such as Bax, Bcl-2, Phospho-Bcl-2, Caspase-3, and Cleaved-Caspase-3, among others. Here, BCL2 is linked to hepatocellular carcinoma.