Compromised immunity following corticosteroid therapy, long-term hospitalization, mechanical ventilation, lactic acidosis, increased ferritin, development of pseudodiabetic conditions due to increased expression of angiotensin-converting enzyme 2 receptor in pancreatic islets, increased insulin resistance due to cytokine storm, and vascular endothelial destruction due to COVID-19 all contribute to an increased mortality rate in patients with simultaneous mucormycosis and COVID-19 [17, 19]. Here, INS is linked to COVID-19.