AIF1 and Alzheimer disease: Microgliosis, a hallmark of AD pathology, was also significantly attenuated: the number of activated microglia in the subiculum was reduced by 53% (Figure S5F, Supporting Information), and microglial phagocytic activity increased by 74%, as demonstrated by colocalization of Iba1‐positive microglia with 6E10‐positive Aβ plaques[46, 47] (Figure S5G, Supporting Information).