To further evaluate the tumorigenic potential of METTL10, we isolated primary gastric tumor cells, referred to as ATK cells, from Cldn18‐CreERT2; Apcfl/fl; Trp53fl/fl; KrasG12D (Cldn18‐ATK) mice.[17] We then generated METTL10‐overexpressing ATK cells (Figure S1s, Supporting Information) and established orthotopic GC models by injecting these cells into the subserosal layer of the stomach. This evidence concerns the gene EEF1AKMT2 and gastric neoplasm.