In contrast, in the COVID‐19 lung at autopsy there were no detectable viral RNA transcripts but ongoing inflammation with increased factors such as TNF and IL‐6, increased Th1 cytokines typically associated with pro‐inflammatory chemotaxis such as CXCL10 and CCL2, and innate antiviral responses such as MX1.72 This evidence concerns the gene CCL2 and COVID-19.