Preclinical studies have demonstrated that cannabinoids can exert cytotoxic effects on cervical cancer cells through mechanisms such as oxidative stress induction, modulation of key signalling pathways such as; phosphoinositide 3-kinase/protein kinase B/mechanistic target of rapamycin (PI3K/AKT/mTOR) pathway, mitogen-activated protein kinase (MAPK)/extracellular signal-regulated kinase (ERK) pathway and caspase pathway, and alteration of immune responses (Pagano et al. 2021). Here, MTOR is linked to cervical cancer.