Furthermore, the addition of anti-α-toxin antibody (MEDI4893*) completely mitigated the NP796- and α-toxin-induced CX3CL1 release in the lung model supernatants (Fig. 2F), suggesting that CX3CL1 released in S. aureus respiratory tract infection is driven by α-toxin. This evidence concerns the gene CX3CL1 and Respiratory tract infection.