PDCD1LG2 and asthma: Fn1 (tissue repair, fibrosis), Arg1 (immune modulation, tissue repair), and Pdcd1lg2 (immune regulation/tolerance), which are upregulated in lung inflammation and asthma signatures [24, 25, 26, 27], showed significantly reduced expression on gene level in Mcpt8Cre mice compared to WT upon lung challenge, indicative of reduced inflammation in basophil‐deficient mice.