Previous reports identified autophagopathies, such as WDR45 defects, as the cause of both childhood NDD41 and early adulthood parkinsonism,42 suggesting substantial clinico‐pathological overlap reflective of the molecular interactions between EPG5 and WDR45 in the autophagosome–lysosome fusion machinery. The gene discussed is WDR45; the disease is Parkinsonism.