FMO2 and myocardial infarction: Flavin‐containing monooxygenase 2 (FMO2), which uses NADP+ and FAD as coenzymes and cofactors to catalyze the oxidative metabolism of many xenobiotics,[19] was suggested to exert a strong anti‐fibrotic effect after myocardial infarction (MI) in our previous study.[20] What raises much interest is that FMO2 is expressed in ECs at a considerable level and conveys a significant reduction under ischemic conditions from a single‐cell dataset.