Importantly, the evaluation of the levels of ATP synthase β subunit (ATPB) and Voltage-dependent anion channel (VDAC), mitochondrial proteins, both overexpressed in glioblastoma and linked to tumor growth and poor prognosis [22, 23], show their significant modulation among the experimental conditions (Fig. 5D). This evidence concerns the gene VDAC1 and glioblastoma.