Our findings are in line with an earlier report where healers versus non‐healers of human diabetic foot ulcers (DFU) were analysed.[12] Enrichment of macrophages overexpressing inflammatory genes, including IL‐1β, S100A8, and S100A9 in DFU‐healers was observed to stimulate an acute inflammatory response to promote wound healing (Figure S9C,D, Supporting Information, derived from publicly available data sets [12]). Here, IL1B is linked to diabetic foot.