The interaction between CAMK2A and miR-3200-3p has been demonstrated to regulate the progression of glioma through the Rat Sarcoma viral oncogene homolog (Ras)/Rapidly Accelerated Fibrosarcoma (Raf)/Mitogen-activated protein kinase kinase (MEK)/Extracellular signal-regulated kinase (ERK) signaling pathway [16]. Here, CAMK2A is linked to central nervous system cancer.