DCAF12 and neoplasm: scRNA‐seq analysis of the GSE149655 dataset demonstrated cell type‐specific dysregulation, with tumor epithelial cells exhibiting the most significant upregulation of DCAF12 compared to their normal counterparts (adjusted p < 0.001; Figure S21A–C, Supporting Information), suggesting that tumor cell‐autonomous mechanisms drive DCAF12 dysregulation during malignant progression.