CACNA1D and lung adenocarcinoma: Using the PR8 (H1N1) IAV isolate, the authors demonstrated, through a series of well‐thought‐out experiments, that IAV infection reduces LTCC‐mediated Ca2+ influx; in human lung adenocarcinoma A549 cells, Cav1.3 is the predominant LTCC isoform, and its knockdown (KD) phenocopies LTCC blockade, consistent with suppression of Cav1.3 activity during IAV infection.